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  • Produktbild: The Year in Endocrinology, 1975–1976
  • Produktbild: The Year in Endocrinology, 1975–1976

The Year in Endocrinology, 1975–1976

Fr. 73.90

inkl. gesetzl. MwSt., Versandkostenfrei

Beschreibung

Produktdetails

Einband

Taschenbuch

Erscheinungsdatum

05.02.2012

Abbildungen

XVIII, 3 illus., schwarz-weiss Illustrationen

Herausgeber

S. Ingbar

Verlag

Springer Us

Seitenzahl

328

Maße (L/B/H)

22.9/15.2/1.9 cm

Gewicht

505 g

Auflage

Softcover reprint of the original 1st edition 1976

Sprache

Englisch

ISBN

978-1-4615-8845-0

Beschreibung

Produktdetails

Einband

Taschenbuch

Erscheinungsdatum

05.02.2012

Abbildungen

XVIII, 3 illus., schwarz-weiss Illustrationen

Herausgeber

S. Ingbar

Verlag

Springer Us

Seitenzahl

328

Maße (L/B/H)

22.9/15.2/1.9 cm

Gewicht

505 g

Auflage

Softcover reprint of the original 1st edition 1976

Sprache

Englisch

ISBN

978-1-4615-8845-0

Herstelleradresse

Springer-Verlag KG
Sachsenplatz 4-6
1201 Wien
AT

Email: ProductSafety@springernature.com

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  • Produktbild: The Year in Endocrinology, 1975–1976
  • Produktbild: The Year in Endocrinology, 1975–1976
  • 1 Neuroendocrinology.- 1.1. Introduction.- 1.2. Neural Regulation of Endocrine Function.- 1.2.1. Characterization of CNS Factors Acting on the Anterior Pituitary Gland.- 1.2.2. Anatomical Localization of Hypophysiotropic Factors.- 1.2.2.1. Thyrotropin-Releasing Factor.- 1.2.2.2. Gonadotropin-Releasing Factor.- 1.2.2.3. Somatostatin.- 1.2.2.4. Neurotransmitter Localization.- 1.2.3. Neurotransmitter Regulation of Releasing Factors.- 1.2.3.1. Corticotropin-Releasing Factor.- 1.2.3.2. Gonadotropin-Releasing Factor.- 1.2.3.3. Prolactin.- 1.2.3.4. Thyrotropin-Releasing Factor.- 1.2.4. Role of Prostaglandins in the Regulation of Hypophysiotropic Factors and Pituitary Hormones.- 1.2.5. Brain and Pituitary Growth Factors.- 1.3. Hormone-Nervous System Interactions.- 1.3.1. Catechol Estrogens.- 1.3.2. CNS Uptake of Steroids.- 1.3.2.1. Corticosteroids.- 1.3.2.2. Androgens.- 1.3.3. Peptide Hormone-Nervous System Interactions.- 1.3.3.1. Effect of Peptide Fragments on Learning and Memory.- 1.3.3.2. Effect of Peptides on Opiate Receptors.- 1.3.3.3. Effect of Releasing Factors on Brain Excitability and behavior.- 1.4. Releasing Factors.- 1.4.1. Interrelationships.- 1.4.2. Therapeutic Uses of Gonadotropin-Releasing Factor.- 1.4.2.1. Anovulation.- 1.4.2.2. Hypogonadism.- 1.5. Neuroendocrine Disease.- 1.5.1. Acromegaly.- 1.5.1.1. Anomalous Responses to Testing.- 1.5.1.2. Response to Medical Therapy.- 1.5.2. Galactorrhea.- 1.5.3. Cushing’s Disease.- 1.5.4. Anorexia Nervosa.- 1.6. The Pineal Gland.- 1.6.1. Regulation of Gonadotropic Function.- 1.6.2. Neurotransmitter-Receptor-Enzyme Interactions.- References.- 2 Anterior Pituitary.- 2.1. Introduction.- 2.2. Corticotropin and Melanocyte-Stimulating Peptides.- 2.3. Thyrotropin.- 2.4. Gonadotropins.- 2.4.1. Plasma and Urine Gonadotropins.- 2.4.2. Gonadotropins in Mother and Fetus.- 2.4.3. Gonadotropins in Children.- 2.4.4. Gonadotropin Responses to Luteinizing Hormone-Releasing Hormone.- 2.5. Prolactin.- 2.6. Somatotropin.- 2.6.1. Chemistry.- 2.6.2. Regulation of Secretion.- 2.6.3. Effects on Carbohydrate Metabolism.- 2.6.4. New Therapeutic Applications.- 2.6.5. Acromegaly.- 2.6.6. Hypopituitarism.- References.- 3 The Thyroid.- 3.1. Introduction.- 3.2. Neuroendocrine Regulation of Thyroid Function.- 3.3. Thyroid Hormones.- 3.3.1. Synthesis and Secretion.- 3.3.2. Peripheral Metabolism.- 3.3.3. Metabolic Action.- 3.4. Clinical Aspects of Thyroidology.- 3.4.1. Thyrotoxicosis.- 3.4.2. Hypothyroidism.- 3.4.3. Thyroid Cancer.- 3.5. Miscellaneous Aspects of Thyroidology.- References.- 4 The Ovary.- 4.1. Introduction.- 4.2. Estrogen Secretion Rates.- 4.2.1. Normal Women.- 4.2.2. Anovulatory Bleeding.- 4.2.3. Hyperthyroidism.- 4.2.4. Oral Estrogens.- 4.2.5. Catechol Estrogens.- 4.2.5.1. Estriol.- 4.3. Regulation of Follicular Growth.- 4.3.1. Intrafollicular Steroid Concentrations.- 4.3.2. Protein Hormones in the Follicles.- 4.3.3. Prostaglandins.- 4.4. The Postmenopausal Ovary.- 4.4.1. Follicle-Stimulating Hormone Regulation.- 4.4.2. Androgen Secretion.- 4.5. Idiopathic Hirsutism.- 4.5.1. The Ovary.- 4.5.2. Therapy.- 4.5.3. Antiandrogens.- 4.5.4. Skin Androgen Metabolism.- 4.6. Side Effects of Estrogen.- 4.6.1. Liver Tumors.- 4.6.2. Gall Bladder Disease.- 4.6.3. Uterine Cancer.- 4.6.4. Stroke.- References.- 5 The Testis.- 5.1. Introduction.- 5.2. Intrauterine and Neonatal Function.- 5.2.1. Embryonic Sex Differentiation.- 5.2.1.1. Normal Controls.- 5.2.1.2. Defects in Testicular Function in Utero.- 5.2.2. Testicular Function in Childhood.- 5.3. Puberty.- 5.4. Adult Physiology.- 5.4.1. The Hypothalamus and Pituitary.- 5.4.2. The Testis.- 5.4.2.1. Steroidogenesis.- 5.4.2.2. Spermatogenesis.- 5.4.2.3. Effect of Age on Human Testicular Function.- 5.5. Adult Pathophysiology.- 5.5.1. Gonadotropin Deficiencies.- 5.5.2. The Testis.- 5.5.2.1. Steroidogenesis.- 5.5.2.2. Tubular Defects.- 5.6. Influence of Other Disease on Testicular Function.- 5.6.1. Uremia.- 5.6.2. Cirrhosis.- 5.7. Conclusions.- References.- 6 The Adrenal Cortex.- 6.1. Introduction.- 6.2. Corticosteroid Biosynthesis.- 6.3. Mechanism of Action of ACTH.- 6.4. Diseases of the Adrenal Cortex.- 6.4.1. Enzymatic Defects in Steroidogenesis.- 6.4.2. Adrenal Hypofunction.- 6.4.3. Cushing’s Syndrome.- 6.4.4. Adrenocortical Neoplasms.- References.- 7 Aldosterone and the Renin-Angiotensin System.- 7.1. Introduction.- 7.2. Increased Aldosterone Production.- 7.2.1. Renin Suppressed.- 7.2.1.1. Syndrome of Primary Hyperaldosteronism.- 7.2.1.2. Location of Adrenocortical Adenoma with [131I] 19-Iodocholesterol.- 7.2.2. Renin Increased.- 7.2.2.1. Malignant or Accelerated Hypertension.- 7.2.2.2. Unilateral Renovascular Disease.- 7.2.2.3. Renin-Secreting Tumors.- 7.2.2.4. “Banter’s Syndrome”.- 7.3. Normal Aldosterone Production—Low-Renin Hypertension.- 7.4. Reduced Aldosterone Production.- 7.4.1. Renin Suppressed.- 7.4.1.1. Hyporeninemic Hypoaldosteronism.- References.- 8 Catecholamines and the Sympathoadrenal System.- 8.1. Introduction.- 8.2. Familial Pheochromocytoma and Multiple Endocrine Adenomatosis (MEA).- 8.2.1. Incidence and Importance.- 8.2.2. Sipple’s Syndrome (MEA-II).- 8.2.2.1. Background.- 8.2.2.2. Presentation.- 8.2.2.3. Pheochromocytoma in MEA-II.- 8.2.2.4. Medullary Carcinoma of the Thyroid.- 8.2.2.5. Hyperparathyroidism.- 8.2.2.6. Management of Patients and Families with MEA-II (Sipple’s Syndrome).- 8.2.3. Mucosal Neuroma Syndrome (MEA-III).- 8.2.4. Multiple Endocrine Adenomatosis—Theories of Pathogenesis.- 8.2.4.1. The APUD Cell Concept.- 8.2.4.2. Nerve Growth Factor.- 8.2.4.3. Nesidioblastosis.- 8.3. Clinical Assessment of the Functional State of the Adrenal Medulla and the Sympathetic Nervous System.- 8.3.1. The Nature of the Problem.- 8.3.2. Assay of Catecholamines in Plasma and Urine.- 8.3.2.1. Plasma Catecholamines.- 8.3.2.2. Urinary Catecholamines.- 8.3.3. Dopamine-?-Hydroxylase Activity in Plasma— A Measure of Sympathetic Activity?.- 8.3.3.1. The Enzyme Dopamine-?-Hydroxylase (DBH).- 8.3.3.2. DBH Activity as a Measure of Sympathetic Activity.- 8.4. Hyperthyroidism and the Sympathoadrenal System.- 8.4.1. Effect of Thyroid Hormones on Catecholamine Biosynthesis, Metabolism, and Excretion.- 8.4.2. Effect of Thyroid Hormones on Sensitivity to Catecholamines.- 8.4.2.1. Thyroid Hormones and the Cardiovascular Effects of Catecholamines.- 8.4.2.2. Metabolic and Calorigenic Effects of Catecholamines in Relation to Thyroid Status.- 8.4.3. Adrenergic Blocking Agents in the Treatment of Hyperthyroidism.- 8.4.3.1. The Choice of an Antiadrenergic Agent.- 8.4.3.2. Effects of Adrenergic Blocking Agents in Hyperthyroidism.- 8.4.3.3. Clinical Usefulness of Adrenergic Blocking Agents in Thyrotoxicosis.- 8.4.3.4. An Open Question: Why Are Adrenergic Blocking Agents Efficacious in the Symptomatic Treatment of Hyperthyroidism?.- References.- 9 Calcitonin.- 9.1. Introduction.- 9.2. Calcitonin.- References.- 10 Ectopic Hormone Syndromes.- 10.1. Introduction.- 10.2. Etiology and Mechanisms of Ectopic Hormone Production..- 10.3. Characteristics of the Ectopic Hormone Syndromes.- 10.4. Recent Developments in Specific Hormone Syndromes.- 10.4.1. Ectopic Production of Corticotropin (ACTH).- 10.4.2. Ectopic Production of Melanocyte-Stimulating Hormone (MSH).- 10.4.3. Ectopic Production of Growth Hormone.- 10.4.4. Ectopic Production of Human Placental Lactogen (HPL) or Human Chorionic Somatomammotropin (HCS).- 10.4.5. Ectopic Production of Prolactin.- 10.4.6. Ectopic Production of Gonadotropins.- 10.4.7. Ectopic Production of Thyrotropin.- 10.4.8. Ectopic Production of Antidiuretic Hormone (ADH).- 10.4.9. Hypoglycemic Syndromes and Tumors.- 10.4.10. Ectopic Production of Parathyroid Hormone (PTH) and Other Calcium-Mobilizing Substances.- 10.4.11. Ectopic Production of Other Hormones.- 10.4.11.1. Calcitonin.- 10.4.11.2. Erythropoietin.- 10.4.11.3. Renin-Secreting Tumors.- 10.4.11.4. Other Prostaglandins.- 10.4.11.5. Osteomalacia, Hypophosphatemia, and Hyperphosphaturia.- 10.4.11.6. Glucagon and Skin Disease; Vasoactive Intestinal Peptide (VIP).- 10.5. Production of Nonhormonal Proteins and Other Syndromes by Malignant Disorders.- 10.5.1. Ectopic Production of Other Proteins.- 10.5.2. Tumor Syndromes Not Known to Be Associated with Definite Humoral Substances.- References.- 11 Current Concepts in Steroid Hormone Action.- 11.1. Introduction.- 11.2. Autoradiography.- 11.3. Receptor-Mediated Steroid Translocation to Nuclei.- 11.4. Quantitation of Nuclear Receptors by Nuclear Exchange..- 11.5. Cell-Free Binding Studies.- 11.6. Nature of the Nuclear Acceptor Site.- 11.7. Hormone Dependency of Nuclear Binding Receptors.- 11.8. Receptor Activation for Nuclear Binding.- 11.9. Correlation of Nuclear Binding with Biological Response.- 11.10. Cell Genetic Variants in Hormone Response.- 11.11. Quantitation of Transcriptional Events in Vitro.- 11.12. Gene Expression in the Chick Oviduct Model System.- 11.13. In Vivo Steroid-Induced Alterations in Chromatin Transcription Assayed in Vitro.- 11.14. Purification and Characterization of Chick Oviduct Progesterone Receptor.- 11.15. In Vitro Studies of Receptor-Mediated Alterations in Chromatin Transcription.- 11.16. Induction of Specific Gene Sequences by Progesterone Receptors in Vitro.- 11.17. Relationship of the Progesterone Receptor Subunit Structure to Its Effects on Differential Gene Expression.- 11.18. A Model for Steroid Hormone Action.- 11.19. Medical Relevance.- References.